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Access to the article is free, however registration and sign-in are required. In response to genotoxic stresses that damage DNA, cells halt at a series of checkpoints during the cell cycle to provide sufficient time for the DNA to be repaired. In a Perspective, Sagata discusses a new study published elsewhere that shows how the kinase Chk1 contributes to S and G2 checkpoints by phosphorylating a key checkpoint phosphatase, Cdc25A.
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