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Description:

Ca2+/calmodulin-dependent protein kinase II (CaMKII) and actin are two crucial molecules involved in long-term potentiation (LTP). In addition to its signaling function, CaMKII plays a structural role via direct interaction with actin filaments, thus coupling functional and structural plasticity in dendritic spines. The status of F-actin, regulated by CaMKII, determines the postsynaptic protein binding capacity and thus may act as a synaptic tag that consolidates LTP.

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      Keywords:

      Synapse,Actin filament,oai:nsdl.org:2200/20110722024235223T,NSDL_SetSpec_BEN,Technology,Content/background information,Geoscience,Dendrite,Amino Acid,Life Science,Magnetic resonance imaging,Chemistry,Physics,Physiology,NSDL

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      English

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      Public - Available to anyone

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      Creative Commons Attribution Non-Commercial Share Alike

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